PMOS · PCOS

By Dr. Mélanie DesChâtelets, ND · Naturopathic Doctor and Menopause Society Certified Practitioner · Burnaby and virtual care across BC

Are you trying to figure out what type of PMOS (formerly PCOS) you have? Adrenal? Inflammatory? Wait, no… post-pill? Insulin-resistant? Maybe you see yourself in a few of the buckets at once and you’re trying to work out how to split yourself between them. A lot of people get stuck right here, because it feels like everyone else has it figured out and knows exactly which bucket they’re in. So let’s talk about PCOS types: where they come from, why they sound so legit, and why they often aren’t as helpful as they seem.

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What do people mean by PCOS types?

Here’s the first source of confusion: the word “type” is being used in two completely different ways, and they get blurred together all the time. One is the popular subtypes you’ve seen online (adrenal, inflammatory, insulin-resistant, post-pill). The other is the four research phenotypes (you might have seen them as phenotype A, B, C, or D). They are not the same thing, so let’s take them one at a time.

Are the popular subtypes a real diagnosis?

The popular subtypes come from mechanism-based theories. They were a way to explain the different reasons PCOS might show up in someone’s body, and that instinct isn’t a bad one. But they aren’t medical diagnoses, they aren’t used in research papers, and they don’t appear in the clinical guidelines. What happened next is the issue: those ideas got turned into fixed categories, and then we built protocols, supplement stacks, even specific diets and workouts around each bucket.

And here’s the truth, the reason so many people come to me frustrated: PCOS doesn’t fit in a neat little bucket. You can be insulin-resistant and have inflammation. There’s so much overlap that people end up trying to split themselves across buckets and merge different protocols, and it gets messy. If you’ve always struggled to fit into one… congratulations. You just figured out why this doesn’t work ahead of everyone else. The good news is that a lot of the evidence-based suggestions actually help several of the “buckets” at the same time. Things get better together.

What are the four Rotterdam phenotypes?

Now the other meaning of “type.” Because PCOS is diagnosed on meeting two of three criteria, it can genuinely look different from one person to the next, and the phenotypes just describe which diagnostic features are present. This isn’t about mechanisms like insulin or inflammation… it’s about what’s actually there on the diagnostic picture:

• Phenotype A: all three features, irregular cycles, high androgens, and ovarian morphology (elevated AMH or ultrasound).
• Phenotype B: irregular cycles and high androgens, but no ovarian morphology.
• Phenotype C: ovarian morphology and high androgens, but regular cycles.
• Phenotype D: irregular cycles and ovarian morphology, but no high-androgen symptoms.

These groupings are genuinely useful for studying large patterns across big populations, and I’m a proponent of that research. It may tell us whether certain groups carry higher or lower risk of certain things, and that’s a step in the right direction.

Why don’t phenotypes tell you what to do?

Where it misses the mark is when we stretch population data onto the individual person in front of us. Even if something is more likely in one phenotype, it doesn’t mean it can’t happen in another. So if one phenotype is “higher risk” for insulin resistance, you should still be screening for insulin resistance in all the others anyway, because the label isn’t a way to exclude it. That actually mirrors the PCOS guideline and what the brightest minds in PCOS care are saying: screen everyone, then individualize based on what’s showing up for you and what isn’t, because you checked. I’m not saying the mechanisms don’t matter. I just don’t think grouping people by them is as helpful as it sounds.

Where do the labels backfire?

Being told you’re “the adrenal type” or “the inflammatory type” can feel like a lightbulb moment, like something finally makes sense. But what usually comes next is “take these supplements, avoid these foods, do this workout,” all built on the idea of one root cause and a one-size protocol. So what if you don’t fit just one? What if your insulin is high and your stress is off the charts? What if your androgens were high two years ago but have come down since? A fixed type can lock you into a plan that doesn’t evolve with you. I’ve seen someone decide they’re “not insulin-resistant” because they aren’t gaining weight, so they never test it. Or get so afraid of “spiking cortisol” that they avoid the exact workout they love and would actually do consistently. The reality: once you’re diagnosed, PCOS is an enduring condition, but it isn’t frozen in time. It adapts to what you’re doing, and your care should too.

What I use instead of fixed types

This is why I don’t use fixed types. Instead I use something that reflects where your body is right now. I call it the PCOS profile, and it’s not a type. It’s a flexible, science-informed way to look at the whole picture, figure out what’s relevant for you right now, and track what we’re watching so we can notice changes before they get loud. It respects real life, that you have constraints and priorities and can’t do everything at once. So if you have no idea which “type” you are, you don’t need to feel bad. And if you feel really dogmatic that you’re a certain type, it probably just matched an adaptation that’s working for you, and that’s good feedback. I’m not saying throw the bathwater out with the baby (an expression I genuinely don’t understand, by the way). I’m saying don’t assume you live in the same bucket forever. Maybe it’s time to let go of the labels and just do a comprehensive assessment of where you’re actually at.

▶ Want the full walk-through, including all four phenotypes? Watch the complete video here.

This article is educational, not medical advice or a diagnosis. If something here resonates, bring it to your healthcare team, who can look at it through the lens of your individual health.

Main references

1. Teede HJ, et al. Recommendations from the 2023 International Evidence-based Guideline for the Assessment and Management of Polycystic Ovary Syndrome. J Clin Endocrinol Metab. 2023. PubMed

By Dr. Mélanie DesChâtelets, ND · Naturopathic Doctor and Menopause Society Certified Practitioner · Burnaby and virtual care across BC

What if your symptoms look like PMOS (formerly PCOS)… but aren’t? Getting this wrong means every decision after it could be off. Because a real PCOS diagnosis is actually two-sided: you have to meet your age-appropriate inclusion criteria, and you have to rule out PCOS look-alikes first. That second half gets skipped all the time. So let’s talk about the labs that help rule out PCOS mimics before the label gets applied.

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Inclusion vs exclusion: why both matter

Someone once asked me, “Hey, I had polycystic morphology on my ultrasound. Isn’t it obvious?” And that’s just not how the diagnostic process works, for good reason. Inclusion criteria are what must be present, and those change with the age you’re diagnosed. Exclusion criteria are what must be absent to avoid a misdiagnosis, and these don’t change with age. They’re the same for everyone. To actually land a PCOS diagnosis, you need both: your age-appropriate inclusion criteria, and a clean set of exclusion labs.

What are PCOS “mimickers”?

Before we call it PCOS, we have to rule out other things that look like it. I sometimes call these mimickers, because they produce the same symptoms as someone with PCOS. Some are common and routinely tested, like thyroid issues and high prolactin. Others are more rare, and those are the ones I see missed. But the guidelines are clear: there’s a set of mandatory exclusion labs for everyone, because if we skip them, we can miss rare conditions that look like PCOS but aren’t. And those people deserve to be caught and treated for what they actually have, not handed a PCOS label.

Which labs are mandatory to rule out PCOS mimics?

There are four, and they’re recommended for everyone at diagnosis, regardless of age or how clear the symptoms seem:

• TSH (thyroid). Thyroid problems can make cycles irregular. If your cycles are off but it’s the thyroid, then it might not be PCOS.
• Prolactin. High levels can block ovulation, which can also look like altered cycles.
• 17-OH progesterone. This screens for congenital adrenal hyperplasia, one of those rarer look-alikes.
• FSH. This checks the brain-ovary communication.

If those were done and came back normal, great, you met the exclusion criteria. If something came back positive, that’s where more nuance and follow-up belong, with your provider.

What are the conditional, case-by-case checks?

Then there are conditional ones, the case-by-case checks. You don’t need these to get a PCOS diagnosis if you’ve met inclusion and cleared the mandatory exclusion labs. But a thoughtful provider keeps them in mind when extra symptoms point somewhere else. A few examples: Cushing’s, if there are features of cortisol excess. Androgen-secreting tumours, if androgen symptoms come on rapidly or severely. And hypothalamic amenorrhea, where a significant gap between energy in and energy out (think very low body weight, over-exercising, or a stretch of weight loss) leads the body to essentially pause the cycle.

What red flags point away from PCOS?

In PCOS, androgen symptoms are slow and progressive. So sudden or very severe hair growth, or voice changes, are red flags for a different cause of high androgens, and that deserves a closer look. A round “moon” face, a pad of fat at the back of the neck, or purple stretch lines on the abdomen can be red flags for high cortisol and Cushing’s. Very low body weight, an extreme training schedule, or very high stress alongside missing periods can point to hypothalamic amenorrhea. And milk discharge when you’re not breastfeeding, or new headaches and vision changes, can be red flags for a prolactin-related issue. None of these are a diagnosis on their own. They don’t automatically mean something serious is going on… but they can, and that’s exactly why they’re worth flagging quickly, so the right testing gets done and anything that needs attention gets caught.

How do you check your own diagnosis?

Here’s the empowering part. You can pull up your own lab work and see whether the four mandatory labs (TSH, prolactin, 17-OH progesterone, FSH) were actually done during your diagnostic process. This isn’t about interpreting your own results… it’s about walking into your next appointment with a sharper question: “were the exclusion labs done, and what did they show?” That’s the kind of question that gets you a real answer.

▶ Want the full walk-through, including the red-flag clusters? Watch the complete video here.

This article is educational, not medical advice or a diagnosis. If something here resonates, bring it to your healthcare team, who can look at it through the lens of your individual health.

Main references

1. Teede HJ, et al. Recommendations from the 2023 International Evidence-based Guideline for the Assessment and Management of Polycystic Ovary Syndrome. J Clin Endocrinol Metab. 2023. PubMed

By Dr. Mélanie DesChâtelets, ND · Naturopathic Doctor and Menopause Society Certified Practitioner · Burnaby and virtual care across BC

Maybe you were diagnosed with PMOS (formerly PCOS) as a teenager. Or maybe you’re under 19 right now and wondering how PCOS is even diagnosed at your age. Here’s the tricky part: diagnosing PCOS in teens is different. The same rules we use for adults don’t apply, and that trips a lot of people up. So if you’re 19 or under, or you were labeled as a teen and you’re older now, this matters… because you’ll want to make sure you actually would have met the adolescent criteria in the first place.

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Why is PCOS in teens diagnosed differently?

Because a teenage body is still finding its rhythm. In the years right after your first period, the communication between your brain and your ovaries is just getting started, so some things that look like PCOS in teens are actually normal for the stage. That’s also why PCOS can look so different from one person to the next… maybe you were given a diagnosis and you look at a friend with the same label and your symptoms barely overlap. Or you suspect you have it but you’ve never really been told what it should look like. To protect against over-diagnosing during a normal phase, the criteria are stricter before 20.

What are the two adolescent criteria?

In this age group, we don’t have a reliable way to assess ovarian morphology, so ultrasound and AMH are off the table. That means the adult “two out of three” rule becomes a stricter “two out of two.” You need both: irregular cycles (defined very specifically for teens, more on that below) and androgen signs, either symptoms or blood work. If you only have one of the two, it can be considered “PCOS at risk,” as long as other causes for those symptoms have been explored first.

What do high androgens look like in a teen?

First we look for the obvious symptoms: aggressive acne that doesn’t respond well to treatment or is very persistent as you get older, a certain kind of hair thinning where the parting line keeps getting wider, or excess hair on the face, tracking up toward the belly button or down the legs.

Here’s a big clinical pearl, though. In PCOS, high-androgen symptoms tend to come on slow and progressive. If excess facial hair shows up very suddenly and very severely, that can point to other causes, and it deserves a different workup. So if it’s fast, flag it… it’s worth raising clearly with your provider so the right testing gets done.

What counts as an irregular cycle for a teen?

This is the part that’s defined differently than in adults, and it all depends on how many years it’s been since your first period. In the first year, irregular cycles are normal. From one to under three years after your first period, cycles shorter than 21 days or longer than 45 days count as irregular. At three years and beyond, the window tightens to shorter than 21 or longer than 35. And at any point more than a year out from your first period, a single cycle of 90 days or more counts too. The cut-offs loosen early on for a reason… your body is still getting the conversation going.

Why doesn’t an ultrasound count under 19?

You may have had a pelvic ultrasound for other reasons, but for diagnosing PCOS, it isn’t reliable until it’s been about 8 years since your first period (which for many is around age 20). Before that, the ovaries normally show more follicles, so an ultrasound can look “polycystic” when nothing is actually wrong. So if you’ve been hanging your diagnosis on an ultrasound done as a teen, it’s worth reconsidering and checking whether you meet the other two criteria.

What does “PCOS at risk” mean, and when do you reassess?

Some teens have a few PCOS features but not quite enough for a clear diagnosis… maybe some irregular cycles but not much on the androgen side, or the reverse. Once other causes have been ruled out, that’s when we’d call it “PCOS at risk.” It’s not a “no,” and it’s not a “yes.” It’s a “let’s keep an eye on this.” The recommendation is to reassess for the first time once it’s been at least 8 years since your first period (which for many is around age 20), when ovarian morphology testing (ultrasound, or AMH if you’d rather not do an internal ultrasound) finally becomes accurate.

I spend a lot of time on this because clarity is everything. If you don’t get this first part right, it affects every decision after it. So whether the answer is a clear yes, a not-yet, or a “let’s watch,” knowing exactly where you stand is the whole point.

▶ Want the full walk-through, with all the age cut-offs explained? Watch the complete video here.

This article is educational, not medical advice or a diagnosis. If something here resonates, bring it to your healthcare team, who can look at it through the lens of your individual health.

Main references

1. Teede HJ, et al. Recommendations from the 2023 International Evidence-based Guideline for the Assessment and Management of Polycystic Ovary Syndrome. J Clin Endocrinol Metab. 2023. PubMed

By Dr. Mélanie DesChâtelets, ND · Naturopathic Doctor and Menopause Society Certified Practitioner · Burnaby and virtual care across BC

Do I really have PMOS (formerly PCOS)… or not? It’s one of the most common questions I hear. Sometimes you’re convinced you have it, but no one has ever actually diagnosed you. Other times you’ve been told you have it, and it just doesn’t feel right, or it looks nothing like the PCOS your friend has. And that’s the tricky part. A PCOS diagnosis isn’t as straightforward as it sounds, and if it isn’t done right, every decision after it can be off track. So here’s exactly how PCOS is diagnosed when you’re 20 or older.

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How is PCOS diagnosed in women 20 and older?

For adults, a PCOS diagnosis comes down to the Rotterdam criteria… the “two out of three” rule. You meet it when two of these three are present, after other causes have been ruled out: signs of high androgens, irregular cycles, or polycystic ovarian morphology (seen on ultrasound, or as an elevated AMH on blood work). Two of three. Not one. One quick nuance on the age line, though: what actually matters isn’t your birthday, it’s that it’s been about 8 years since your first period (which for most people is around age 20). Before that, the diagnostic rules are different. The ruling-out-other-causes part is its own important step, and a whole separate conversation. But the inclusion side starts here.

What counts as high androgens?

High androgens can show up as symptoms or on labs. The symptoms look like excess hair growth on the face, or tracking down toward the belly button or the thighs. Persistent acne that doesn’t let up after the teen years, or acne that’s more aggressive and treatment-resistant when you’re younger. Or female-pattern hair thinning, usually at the crown, where the parting line slowly widens.

And here’s the important part. If those symptoms are clearly present, that alone can count. You don’t always need blood work to confirm high androgens, because we can see them. When the symptoms are subtle or unclear, that’s when labs help. The main ones are total and free testosterone, and when free testosterone isn’t available, we can calculate it using the free androgen index. Sometimes we also look at DHEA-S, which comes from the adrenal glands but is still very androgenic.

Why can’t you test androgens while you’re on the pill?

Here’s a major quick pearl. If you’re on the combined oral contraceptive pill, it’s really hard to reliably measure your androgens. The pill raises sex hormone binding globulin and lowers your available androgens. So if your androgens were tested while you were on the pill, you can’t rely on those numbers. If testing is truly necessary, the guidelines suggest coming off the pill for at least three months, using another form of contraception during that time, and then testing.

What actually counts as an irregular cycle?

This one is defined pretty specifically… it’s not just being off by a couple of days. Irregular means cycles shorter than 21 days or longer than 35 days, or fewer than eight cycles a year. And if you’re more than a year past your first period and you’ve had a single cycle stretch past 90 days, that counts as irregular too.

Do you need an ultrasound to diagnose PCOS?

Not always. This is where the myths pile up. Myth one: everyone needs a pelvic ultrasound. Not true. If you already meet two of the three criteria, you don’t need one. Myth two: an ultrasound done as a teenager still counts. It doesn’t. A pelvic ultrasound isn’t reliable for diagnosing PCOS until it’s been about 8 years since your first period (which for most is around age 20). Before that, the ovaries normally show more follicles, so the scan can look “polycystic” when nothing is actually wrong. Myth three, the big one: polycystic ovarian morphology means ovarian cysts. It doesn’t. What we’re actually counting is follicles. In plain language, think of it as how many eggs are running the show. That’s a completely different thing from ovarian cysts.

And here’s the 2023 update worth knowing about. There’s now a blood test, AMH, that can stand in for the ultrasound. If AMH is elevated, it correlates with a high follicle count. This matters because not everyone has access to a pelvic ultrasound, and some people don’t feel comfortable with an internal one. So it opens a door to access.

Can one symptom on its own mean PCOS?

No. An irregular period on its own isn’t PCOS. High-androgen symptoms on their own aren’t PCOS. One ultrasound finding on its own isn’t PCOS. You need two of the three criteria, and you need the other causes ruled out. And if you’re 19 or younger, the diagnostic rules shift… that’s its own video.

Because here’s the thing I keep coming back to: with clarity comes power. When you actually understand what your PCOS diagnosis rests on, every decision after it gets a little easier… and a lot less frustrating.

▶ Want the full walk-through, with the examples and the nuance? Watch the complete video here.

This article is educational, not medical advice or a diagnosis. If something here resonates, bring it to your healthcare team, who can look at it through the lens of your individual health.

Main references

1. Teede HJ, et al. Recommendations from the 2023 International Evidence-based Guideline for the Assessment and Management of Polycystic Ovary Syndrome. J Clin Endocrinol Metab. 2023. PubMed

By Dr. Mélanie DesChâtelets, ND · Naturopathic Doctor and Menopause Society Certified Practitioner · Burnaby and virtual care across BC

Have you heard someone say you can reverse PCOS, or maybe even cure it? I want to talk about why that messaging is misleading, and honestly, why I think it’s a little dangerous. PMOS (formerly PCOS) is what the clinical guidelines call an enduring diagnosis. If you’ve been properly diagnosed, it’s a diagnosis you have for life. So “cure” and “reverse” set you up to expect something that isn’t how this condition works. And the cost of that expectation is real.

I’m not saying this to take anything away from you. I’m saying it because the truth gives you better footing than a promise that doesn’t hold.

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Can you cure PCOS?

No. PMOS · PCOS is an enduring diagnosis, and that’s not my opinion, it’s the consensus in the clinical guidelines. The 2023 international PCOS guideline treats it as a condition you carry for life once you’ve been accurately diagnosed.

Part of why matters here. PMOS isn’t a simple diagnosis. With a lot of conditions, one lab flags and you have the diagnosis. PMOS · PCOS has inclusion criteria (which change depending on your age at diagnosis) and exclusion criteria. More moving parts means more room for error, so completing the diagnostic process properly is the thing that matters. If you’re not sure yours was done thoroughly, that’s a good conversation to bring to your own healthcare team.

Can you reverse PCOS naturally?

You can change how your symptoms show up, sometimes dramatically. You can’t reverse the diagnosis itself. Those are two different things, and the difference is the whole point.

Here’s the good news. Lifestyle, and sometimes supplements or medication, can have a pretty potent impact on how symptoms present. Someone who’s very symptomatic at diagnosis can do real work and no longer have those symptoms. That’s worth celebrating. It just doesn’t undo the diagnosis underneath.

I’ll be honest. I got this wrong early in my career. I thought if you no longer met the diagnostic criteria, you no longer had it. Like high cholesterol: you do the bloodwork, you change some things, your cholesterol normalizes, and the diagnosis drops off. That’s a real thing for cholesterol. It is not how PMOS · PCOS works.

If my symptoms are gone, do I still have PCOS?

Yes. Feeling good and looking good on the surface doesn’t mean the underlying condition left.

The reasons come down to a few things:

• The cause is multifactorial. Genetics is one driver, and you can’t just turn that off.
• Even when you feel better, people with PMOS · PCOS carry higher health risks that still need monitoring compared to people without it.
• That higher risk holds even when two people are matched for body size. Same size, same everything, the one difference is the PMOS diagnosis, and the risk is still higher.

I want to be clear this isn’t me trying to scare you. Higher risk of something doesn’t mean you’ll get it. It just means it belongs on your radar so you can keep an eye on it with your care team. The problem comes when we pretend the diagnosis is gone and stop checking. That’s how things get missed.

Why does PCOS need ongoing monitoring?

Because the real harm of “cure” language is that people stop watching for the things worth catching early.

“Cure” gives the illusion that a certain set of actions will manage everyone’s symptoms the same way. So someone takes positive action, it improves their symptoms to a point, and then there are leftover symptoms that still need managing in other ways. With “cure” framing, that feels like failure. It isn’t. It’s a reflection of how complex this is and how much genetics is underneath it.

If you believe you’ve reversed it, the quiet risk is that you drop the monitoring. Then a risk that could have been caught early gets missed. That’s the danger I keep coming back to. Not the word itself, but what the word makes people stop doing.

Do I need repeat ultrasounds to track PCOS?

No. We don’t monitor PMOS · PCOS with more and more ultrasounds.

That doesn’t mean you’ll never have another ultrasound. It means the reason for one should be something other than “tracking my PCOS.” So how do you know you’re heading in the right direction? Your symptoms tell you more than the ultrasound does. If your cycles improve, we’d actually expect the ovary picture to improve too, because the communication between your brain and your ovaries is healthier. The way your ovaries look on a scan (morphology is just the fancy word for it) follows the cycles, not the other way around.

What should I focus on instead of a cure?

Focus on sustainable systems over willpower. Not a finish line you sprint to and then you’re done.

Think of it like a dimmer switch, not a light switch. This isn’t a light we turn off. It’s a dimmer. There are real things you can do to make the symptoms less bright. For some people that goes as far as cycles normalizing, less acne, fewer androgenic symptoms, less hair loss on the head and less hair growth on the face. The lights can get a lot dimmer. They don’t switch off.

So don’t treat this as a mountain to conquer once. It’s for life, and that doesn’t mean there’s nothing you can do to feel better than you do today. It means we shift the goal away from perfectionism and toward something personalized to you. Accuracy matters here, because the cost of inaccuracy isn’t abstract: it’s not getting the care you need, not being monitored, and risks going unspotted.

Want the full walk-through in my own words? Watch the full video here, and if it lands, subscribe to my YouTube channel for more like it.

This article is educational, not medical advice or a diagnosis. If something here resonates, bring it to your healthcare team, who can look at it through the lens of your individual health.

Main references

1. Teede HJ, et al. Recommendations from the 2023 International Evidence-based Guideline for the Assessment and Management of Polycystic Ovary Syndrome. J Clin Endocrinol Metab. 2023. PubMed